Na+-H+ exchanger inhibition : A new antihypertrophic tool
Cardiac hypertrophy (CH) is a major risk factor for cardiac death and commonly precedes the development of heart failure (HF). This is motivating the search for novel pharmacological strategies to prevent the development and/or regress CH. Although the signaling pathways leading to myocardial hypert...
Guardado en:
| Autores principales: | Cingolani, Horacio Eugenio, Camilión de Hurtado, María Cristina |
|---|---|
| Formato: | Articulo |
| Lenguaje: | Inglés |
| Publicado: |
2002
|
| Materias: | |
| Acceso en línea: | http://sedici.unlp.edu.ar/handle/10915/84978 |
| Aporte de: |
Ejemplares similares
-
Regression of cardiomyocyte hypertrophy in SHR following chronic inhibition of the Na+/H+ exchanger
por: Camilión de Hurtado, María Cristina, et al.
Publicado: (2002) -
Regression of isoproterenol-induced cardiac hypertrophy by Na+/H+ exchanger inhibition
por: Ennis, Irene Lucía, et al.
Publicado: (2003) -
Thioredoxin 1 (TRX1) Overexpression Cancels the Slow Force Response (SFR) Development
por: Zavala, Maite Raquel, et al.
Publicado: (2021) -
Endogenous endothelin 1 mediates angiotensin II-induced hypertrophy in electrically paced cardiac myocytes through EGFR transactivation, reactive oxygen species and NHE-1
por: Correa, María Verónica, et al.
Publicado: (2014) -
Reduced sarcolemmal expression and function of the NBCe1 isoform of the Na+‒HCO¯3 cotransporter in hypertrophied cardiomyocytes of spontaneously hypertensive rats: Role of the renin-angiotensin system
por: Orlowski, Alejandro, et al.
Publicado: (2014)