Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes

Celiac disease (CD) is a chronic inflammatory disease caused by a genetic predisposition to an abnormal T cell-mediated immune response to the gluten in the diet. Different environmental proinflammatory factors can influence and amplify the T cell-mediated response to gluten. The aim of this manuscr...

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Autores principales: Porpora, Monia, Conte, Mariangela, Lania, Giuliana, Bellomo, Claudia, Rapacciuolo, Luciano, Chirdo, Fernando Gabriel, Auricchio, Renata, Troncone, Riccardo, Auricchio, Salvatore, Barone, Maria Vittoria, Nanayakkara, Merlin
Formato: Articulo
Lenguaje:Inglés
Publicado: 2022
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ERK
Acceso en línea:http://sedici.unlp.edu.ar/handle/10915/156003
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spelling I19-R120-10915-1560032023-08-07T20:07:30Z http://sedici.unlp.edu.ar/handle/10915/156003 Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes Porpora, Monia Conte, Mariangela Lania, Giuliana Bellomo, Claudia Rapacciuolo, Luciano Chirdo, Fernando Gabriel Auricchio, Renata Troncone, Riccardo Auricchio, Salvatore Barone, Maria Vittoria Nanayakkara, Merlin 2022-02 2023-08-07T17:02:43Z en Biología Ciencias Médicas small intestine potential celiac disease NF-κB ERK Celiac disease (CD) is a chronic inflammatory disease caused by a genetic predisposition to an abnormal T cell-mediated immune response to the gluten in the diet. Different environmental proinflammatory factors can influence and amplify the T cell-mediated response to gluten. The aim of this manuscript was to study the role of enterocytes in CD intestinal inflammation and their response to different proinflammatory factors, such as gliadin and viruses. Intestinal biopsies from CD patients on a gluten-containing (GCD-CD) or a gluten-free diet (GFD-CD) as well as biopsies from potential CD patients (Pot-CD) before the onset of intestinal lesions and controls (CTR) were used to investigate IL-1β and IL-6 mRNA levels in situ. Organoids from CD patients were used to test the levels of NF-κB, ERK, IL-6, and IL-1β by Western blot (WB), ELISA, and quantitative PCR. The Toll-like receptor ligand loxoribine (Lox) and gliadin peptide P31-43 were used as proinflammatory stimuli. In CD biopsies inflammation markers IL-1β and IL-6 were increased in the enterocytes, and also in Pot-CD before the onset of the intestinal lesion and in GFD-CD. The inflammatory markers pNF-κB, pERK, IL-1β, and IL-6 were increased and persistent in CD organoids; these organoids were more sensitive to P31-43 and Lox stimuli compared with CTR organoids. Taken together, these observations point to constitutive inflammation in CD enterocytes, which are more sensitive to inflammatory stimuli such as food components and viruses. Instituto de Estudios Inmunológicos y Fisiopatológicos Articulo Articulo http://creativecommons.org/licenses/by/4.0/ Creative Commons Attribution 4.0 International (CC BY 4.0) application/pdf
institution Universidad Nacional de La Plata
institution_str I-19
repository_str R-120
collection SEDICI (UNLP)
language Inglés
topic Biología
Ciencias Médicas
small intestine
potential celiac disease
NF-κB
ERK
spellingShingle Biología
Ciencias Médicas
small intestine
potential celiac disease
NF-κB
ERK
Porpora, Monia
Conte, Mariangela
Lania, Giuliana
Bellomo, Claudia
Rapacciuolo, Luciano
Chirdo, Fernando Gabriel
Auricchio, Renata
Troncone, Riccardo
Auricchio, Salvatore
Barone, Maria Vittoria
Nanayakkara, Merlin
Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes
topic_facet Biología
Ciencias Médicas
small intestine
potential celiac disease
NF-κB
ERK
description Celiac disease (CD) is a chronic inflammatory disease caused by a genetic predisposition to an abnormal T cell-mediated immune response to the gluten in the diet. Different environmental proinflammatory factors can influence and amplify the T cell-mediated response to gluten. The aim of this manuscript was to study the role of enterocytes in CD intestinal inflammation and their response to different proinflammatory factors, such as gliadin and viruses. Intestinal biopsies from CD patients on a gluten-containing (GCD-CD) or a gluten-free diet (GFD-CD) as well as biopsies from potential CD patients (Pot-CD) before the onset of intestinal lesions and controls (CTR) were used to investigate IL-1β and IL-6 mRNA levels in situ. Organoids from CD patients were used to test the levels of NF-κB, ERK, IL-6, and IL-1β by Western blot (WB), ELISA, and quantitative PCR. The Toll-like receptor ligand loxoribine (Lox) and gliadin peptide P31-43 were used as proinflammatory stimuli. In CD biopsies inflammation markers IL-1β and IL-6 were increased in the enterocytes, and also in Pot-CD before the onset of the intestinal lesion and in GFD-CD. The inflammatory markers pNF-κB, pERK, IL-1β, and IL-6 were increased and persistent in CD organoids; these organoids were more sensitive to P31-43 and Lox stimuli compared with CTR organoids. Taken together, these observations point to constitutive inflammation in CD enterocytes, which are more sensitive to inflammatory stimuli such as food components and viruses.
format Articulo
Articulo
author Porpora, Monia
Conte, Mariangela
Lania, Giuliana
Bellomo, Claudia
Rapacciuolo, Luciano
Chirdo, Fernando Gabriel
Auricchio, Renata
Troncone, Riccardo
Auricchio, Salvatore
Barone, Maria Vittoria
Nanayakkara, Merlin
author_facet Porpora, Monia
Conte, Mariangela
Lania, Giuliana
Bellomo, Claudia
Rapacciuolo, Luciano
Chirdo, Fernando Gabriel
Auricchio, Renata
Troncone, Riccardo
Auricchio, Salvatore
Barone, Maria Vittoria
Nanayakkara, Merlin
author_sort Porpora, Monia
title Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes
title_short Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes
title_full Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes
title_fullStr Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes
title_full_unstemmed Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes
title_sort inflammation is present, persistent and more sensitive to proinflammatory triggers in celiac disease enterocytes
publishDate 2022
url http://sedici.unlp.edu.ar/handle/10915/156003
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