PKA-dependent and independent cAMP signaling in 3T3-L1 fibroblasts differentiation

Adipogenesis is stimulated in 3T3-L1 fibroblast by a combination of insulin, dexamethasone, and methylisobutylxanthine (MIX). Mitotic clonal expansion (MCE) precedes differentiation of 3T3-L1 fibroblast to adipocytes. MIX increases cAMP content, which is the activator of protein kinase A (PKA). Howe...

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Autor principal: Martini, C.N
Otros Autores: Plaza, M.V, Vila, M.d.C
Formato: Capítulo de libro
Lenguaje:Inglés
Publicado: 2009
Acceso en línea:Registro en Scopus
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Registro en la Biblioteca Digital
Aporte de:Registro referencial: Solicitar el recurso aquí
LEADER 09983caa a22011057a 4500
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003 AR-BaUEN
005 20230518203850.0
008 190411s2009 xx ||||fo|||| 00| 0 eng|d
024 7 |2 scopus  |a 2-s2.0-57749200703 
024 7 |2 cas  |a bucladesine, 16980-89-5, 362-74-3; cyclic AMP, 60-92-4; n [2 (4 bromocinnamylamino)ethyl] 5 isoquinolinesulfonamide, 127243-85-0; 1-Methyl-3-isobutylxanthine, 28822-58-4; 8-cyclopentyl-1,3-dimethylxanthine, 35873-49-5; Cyclic AMP, 60-92-4; Cyclic AMP-Dependent Protein Kinases, EC 2.7.11.11; Dexamethasone, 50-02-2; H 89, 127243-85-0; Insulin, 11061-68-0; Isoquinolines; Protein Kinase Inhibitors; Sulfonamides; Theophylline, 58-55-9 
040 |a Scopus  |b spa  |c AR-BaUEN  |d AR-BaUEN 
030 |a MCEND 
100 1 |a Martini, C.N. 
245 1 0 |a PKA-dependent and independent cAMP signaling in 3T3-L1 fibroblasts differentiation 
260 |c 2009 
270 1 0 |m Vila, M.d.C.; Departamento de Química Biológica, Facultad de Ciencias Exactas y Naturales, Universidad de Buenos Aires, Pabellon 2, Ciudad Universitaria, 1428 Buenos Aires, Argentina; email: mvila@qb.fcen.uba.ar 
506 |2 openaire  |e Política editorial 
504 |a Bradford, M., A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye-binding (1976) Analytical Biochemistry, 72, pp. 248-254 
504 |a Clegg, C.H., Abrahamsen, M.S., Degen, J.L., Morris, D.R., McKnight, G.S., Cyclic AMP-dependent protein kinase controls basal gene activity and steroidogenesis in Y1 adrenal tumor cells (1992) Biochemistry, 31, pp. 3720-3726 
504 |a Chin, E.C., Abayasekara, D.R.E., Progesterone secretion by luteinizing human granulosa cells: a possible cAMP-dependent but PKA-independent mechanism involved in its regulation (2004) Journal of Endocrinology, 183, pp. 51-60 
504 |a Gregoire, F.M., Smas, C.M., Sul, H.S., Understanding adipocyte differentiation (1998) Physiological Reviews, 78, pp. 783-809 
504 |a Holz, G.G., Kang, G., Harbeck, M., Roe, M.W., Chepurny, O.G., Cell physiology of cAMP sensor Epac (2006) Journal of Physiology, 577 (PART 1), pp. 5-15 
504 |a Jin, S., Zhai, B., Qiu, Z., Wu, J., Lane, M.D., Liao, K., c-Crk, a substrate of the insulin-like growth factor-1 receptor tyrosine kinase, functions as an early signal mediator in the adipocyte differentiation process (2000) Journal of Biological Chemistry, 275, pp. 34344-34352 
504 |a Kanda, Y., Watanabe, Y., Adrenaline increases glucose transport via a Rap1-p38MAPK pathway in rat vascular smooth muscle cells (2007) British Journal of Pharmacology, 151, pp. 476-482 
504 |a Kato, Y., Ozaki, N., Yamada, T., Miura, Y., Oiso, Y., H-89 potentiates adipogenesis in 3T3-L1 cells by activating insulin signaling independently of protein kinase A (2007) Life Science, 80, pp. 476-483 
504 |a Kawasaki, H., Springett, G.M., Mochizuki, N., Toki, S., Nakaya, M., Matsuda, M., Housman, D.E., Graybiel, A.M., A family of cAMP-binding proteins that directly activate Rap1 (1998) Science, 282, pp. 2275-2279 
504 |a Lean, M.E., Pathophysiology of obesity (2000) The Proceedings of the Nutrition Society, 59, pp. 331-336 
504 |a Liao, K., Lane, M.D., The blockade of preadipocyte differentiation by protein-tyrosine phosphatase HA2 is reversed by vanadate (1995) Journal Biological Chemistry, 270, pp. 12123-12132 
504 |a Mellon, P.L., Clegg, C.H., Correll, L.A., McKnight, G.S., Regulation of transcription by cyclic AMP-dependent protein kinase. (1989) Proceedings of the National Academy of Sciences, United States of America, 86, pp. 4887-4891 
504 |a Patel, Y.M., Lane, M.D., Mitotic clonal expansion during preadipocyte differentiation: calpain-mediated turnover of p27 (2000) Journal of Biological Chemistry, 275, pp. 17653-17660 
504 |a Qiu, Z., Wei, Y., Chen, N., Jiang, M., Wu, J., Liao, K., DNA synthesis and mitotic clonal expansion is not a required step for 3T3-L1 preadipocytes into adipocytes (2001) Journal of Biological Chemistry, 276, pp. 11988-11995 
504 |a Rehmann, H., Das, J., Knipscheer, P., Wittinghofer, A., Bos, J.L., Structure of the cyclic-AMP-responsive exchange factor Epac2 in its auto-inhibited state (2006) Nature, 439, pp. 625-628 
504 |a Rubin, C.S., Hirsch, A., Fung, C., Rosen, O.M., Development of hormone receptors and hormonal responsiveness in vitro. Insulin receptors and insulin sensitivity in the preadipocyte and adipocyte forms of 3T3-L1 cells (1978) Journal of Biological Chemistry, 253, pp. 7570-7578 
504 |a Student, A.K., Hsu, R.Y., Lane, M.D., Induction of fatty acid synthetase synthesis in differentiating 3T3-L1 preadipocytes (1980) Journal of Biological Chemistry, 255, pp. 4745-4750 
504 |a Varone, C.L., Giono, L.E., Ochoa, A., Zakin, M.M., Cánepa, E.T., Transcriptional regulation of 5-aminolevulinate synthase by phenobarbital and cAMP-dependent protein kinase (1999) Archives of Biochemistry and Biophysics, 372, pp. 261-270 
504 |a Xu, J., Liao, K., Protein kinase B/AKT 1 plays a pivotal role in insulin-like growth factor-1 receptor signaling induced 3T3-L1 adipocyte differentiation (2004) Journal of Biological Chemistry, 279, pp. 35914-35922 
504 |a Yeh, W., Bierer, B.E., McKnight, S.L., Rapamycin inhibits clonal expansion and adipogenic differentiation of 3T3-L1 cells (1995) Proceedings of the National Academy of Sciences, United States of America, 92, pp. 11086-11090 
504 |a Yin, F., Wang, Y.Y., Du, J.H., Li, C., Lu, Z.Z., Han, C., Zhang, Y.Y., Noncanonical cAMP pathway and p38 MAPK mediate beta2-adrenergic receptor-induced IL-6 production in neonatal mouse cardiac fibroblasts (2006) Journal of Molecular and Cellular Cardiology, 40, pp. 384-393 
520 3 |a Adipogenesis is stimulated in 3T3-L1 fibroblast by a combination of insulin, dexamethasone, and methylisobutylxanthine (MIX). Mitotic clonal expansion (MCE) precedes differentiation of 3T3-L1 fibroblast to adipocytes. MIX increases cAMP content, which is the activator of protein kinase A (PKA). However, PKA-independent cAMP signaling has also been described. In this paper, it was found that H89, an inhibitor of PKA, was able to block MCE but not differentiation of 3T3-L1 fibroblast. Consistently, MCE did not occur in the absence of MIX in the differentiation mixture but was recovered by overexpression of a catalytic subunit of PKA. In addition, the transfection of 3T3-L1 fibroblast with a dominant-negative mutant of PKA inhibited MCE. On the other hand, differentiation of 3T3-L1 fibroblast to adipocytes did not occur when MIX was not present in the differentiation mixture and it could not be recovered by overexpression of a catalytic subunit of PKA. Differentiation was restored by addition of either dibutyryl-cAMP (db-cAMP) or 8 CPT-2 Me-cAMP. The latter activates cAMP-EPAC but not PKA signaling. These results indicate that cAMP-PKA-independent signaling, is required for 3T3-L1 fibroblasts differentiation to adipocytes and MIX signaling through cAMP-PKA is necessary for MCE, although MCE is not essential for adipogenesis. © 2008 Elsevier Ireland Ltd. All rights reserved.  |l eng 
536 |a Detalles de la financiación: Consejo Nacional de Investigaciones Científicas y Técnicas 
536 |a Detalles de la financiación: This work was supported by Consejo Nacional de Investigaciones Científicas y Técnicas and Universidad de Buenos Aires, Argentina. 
593 |a Departamento de Química Biológica, Facultad de Ciencias Exactas y Naturales, Universidad de Buenos Aires, Pabellon 2, Ciudad Universitaria, 1428 Buenos Aires, Argentina 
690 1 0 |a 3T3-L1 FIBROBLAST 
690 1 0 |a ADIPOGENESIS 
690 1 0 |a CAMP 
690 1 0 |a INSULIN 
690 1 0 |a METHYLISOBUTYLXANTHINE (MIX) 
690 1 0 |a MITOTIC CLONAL EXPANSION 
690 1 0 |a PKA 
690 1 0 |a 8 (4 CHLOROPHENYLTHIO) 2' O METHYLADENOSINE 3',5' CYCLIC MONPHOSPHATE 
690 1 0 |a BUCLADESINE 
690 1 0 |a CYCLIC AMP 
690 1 0 |a CYCLIC AMP DEPENDENT PROTEIN KINASE 
690 1 0 |a N [2 (4 BROMOCINNAMYLAMINO)ETHYL] 5 ISOQUINOLINESULFONAMIDE 
690 1 0 |a UNCLASSIFIED DRUG 
690 1 0 |a ADIPOCYTE 
690 1 0 |a ANIMAL CELL 
690 1 0 |a ARTICLE 
690 1 0 |a CATALYSIS 
690 1 0 |a CELL DIFFERENTIATION 
690 1 0 |a FIBROBLAST 
690 1 0 |a GENETIC TRANSFECTION 
690 1 0 |a MOUSE 
690 1 0 |a NONHUMAN 
690 1 0 |a PRIORITY JOURNAL 
690 1 0 |a PROTEIN EXPRESSION 
690 1 0 |a 1-METHYL-3-ISOBUTYLXANTHINE 
690 1 0 |a 3T3-L1 CELLS 
690 1 0 |a ADIPOGENESIS 
690 1 0 |a ANIMALS 
690 1 0 |a CELL DIFFERENTIATION 
690 1 0 |a CYCLIC AMP 
690 1 0 |a CYCLIC AMP-DEPENDENT PROTEIN KINASES 
690 1 0 |a DEXAMETHASONE 
690 1 0 |a FIBROBLASTS 
690 1 0 |a INSULIN 
690 1 0 |a ISOQUINOLINES 
690 1 0 |a MICE 
690 1 0 |a MITOSIS 
690 1 0 |a PROTEIN KINASE INHIBITORS 
690 1 0 |a SIGNAL TRANSDUCTION 
690 1 0 |a SULFONAMIDES 
690 1 0 |a THEOPHYLLINE 
700 1 |a Plaza, M.V. 
700 1 |a Vila, M.d.C. 
773 0 |d 2009  |g v. 298  |h pp. 42-47  |k n. 1-2  |p Mol. Cell. Endocrinol.  |x 03037207  |w (AR-BaUEN)CENRE-6138  |t Molecular and Cellular Endocrinology 
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856 4 0 |u https://doi.org/10.1016/j.mce.2008.10.023  |y DOI 
856 4 0 |u https://hdl.handle.net/20.500.12110/paper_03037207_v298_n1-2_p42_Martini  |y Handle 
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963 |a VARI 
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