Trypanosoma cruzi: Aberrant expression of class II major histocompatibility complex molecules in skeletal and heart muscle cells of chronically infected mice

In isolated skeletal, heart, and smooth muscle cells from BALB/c and C3H/HeJ mice infected with different strains of Trypanosoma cruzi the presence of class II MHC molecules was investigated by immunocytochemical techniques. We employed single muscle fibers instead of conventional cryostat sections...

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Autor principal: Meckert, P.C
Otros Autores: Hontebeyrie-Joskowicz, M., Chambo, J., Levin, M., Laguens, R.P
Formato: Capítulo de libro
Lenguaje:Inglés
Publicado: 1991
Acceso en línea:Registro en Scopus
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LEADER 08741caa a22010457a 4500
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024 7 |2 scopus  |a 2-s2.0-0026101983 
024 7 |2 cas  |a Histocompatibility Antigens Class II 
040 |a Scopus  |b spa  |c AR-BaUEN  |d AR-BaUEN 
030 |a EXPAA 
100 1 |a Meckert, P.C. 
245 1 0 |a Trypanosoma cruzi: Aberrant expression of class II major histocompatibility complex molecules in skeletal and heart muscle cells of chronically infected mice 
260 |c 1991 
270 1 0 |m Laguens, R.P.; Cátedra de Patología II, Facultad de Ciencias Médicas, Universidad de La Plata, Calle 60 y 120, 1900 La Plata, Argentina 
506 |2 openaire  |e Política editorial 
504 |a Andrade, Chagas' disease: Pathology of the cardiac lesions (1976) American Trypanosomiasis Research Pan American Health Organization Scientific Publication 318, pp. 146-151 
504 |a Ben Younes-Chennoufi, Hontebeyrie-Joskowicz, Tricottet, Eisen, Reynes, Said, Persistence of Try-panosoma antigens in inflammatory lesions of chronically infected mice (1988) Transactions of the Royal Society of Tropical Medicine and Hygiene, 82, pp. 77-83 
504 |a Ben Younes-Chennoufi, Said, Eisen, Durand, Hontebeyrie-Joskowicz, Cellular immunity to Trypanosoma cruzi is mediated by helper T cells (CD4+) (1988) Transactions of the Royal Society of Tropical Medicine and Hygiene, 82, pp. 84-89 
504 |a Bottazzo, Dean, McNally, MacKay, Swift, Gamble, In situ characterization of autoimmune phenomena and expression of HLA molecules in the pancreas in diabetic insulitis (1985) New England Journal of Medicine, 313, pp. 353-360 
504 |a Bottazzo, Todd, Mirakian, Belfiore, Pujol Borrel, Organ specific autoimmunity: A 1986 overview (1986) Immunological Reviews, 94, pp. 137-169 
504 |a Cabeza Meckert, Laguens, Selectivity of alterations in skeletal fibers in chronic Chagas' disease in the mouse (1988) Experientia, 44, pp. 14-16 
504 |a Dean, Walker, Bone, Baird, Cooke, Prediabetes in the spontaneously diabetic BB/E rat: Lymphocyte subpopulations in the pancreatic infiltrate and expression of rat MHC class II molecules in endocrine cells (1985) Diabetologia, 28, pp. 464-466 
504 |a Guesdon, Tenynck, Avrameas, The use of avidin biotin interaction in immunoenzymatic technics (1979) Journal of Histochemistry and Cytochemistry, 27, pp. 11-31 
504 |a Hanafusa, Fugino-Kurihara, Myazaki, Yamada, Nakajima, Miyagawa, Tarui, Expression of class II major histocompatibility complex antigens on pancreatic B cells in NOD mouse (1987) Diabetologia, 30, pp. 104-108 
504 |a Hart, Waldschmidt, Hanley-Hyde, Moore, Kemp, Schelper, Brain microvascular smooth muscle express class II antigens (1987) The Journal of Immunology, 138, pp. 2960-2963 
504 |a Hontebeyrie-Joskowicz, Said, Milon, Marchal, Eisen, L3T4+ cells able to mediate parasite-specific delayed type hypersensibility play a role in the pathology of experimental Chagas disease (1987) European Journal of Immunology, 17, pp. 1033-1037 
504 |a Jonasson, Holm, Skalli, Gabbiani, Hansson, Expression of class II transplantation antigen on vascular smooth muscle cells in humans atherosclerosis (1985) Journal of Clinical Investigation, 76, pp. 125-131 
504 |a Laguens, Cabeza Meckert, Gelpi, Chronic Chagas disease in the mouse. I Electrocardiographic and morphological patterns of the cardiopathy (1981) Medicina (Buenos Aires), 41, pp. 35-39 
504 |a Laguens, Cabeza Meckert, Chambo, Gelpi, Chronic Chagas disease in the mouse. II Transfer of the heart disease by means of immunocompetent cells (1981) Medicina (Buenos Aires), 41, pp. 40-43 
504 |a Laguens, Cabeza Meckert, Chambo, Anti-heart antibody-dependent cytotoxicity in the sera of mice chronically infected with Trypanosoma cruzi (1988) Infection and Immunity, 56, pp. 993-997 
504 |a Mosquera, Interferon-γ induces class II antigen expression on cultured rat mesangial smooth muscle cells (1989) Clinical Immunology and Immunopathology, 53, pp. 341-345 
504 |a Petry, Eisen, Chagas disease: A model for the study of autoimmune disease (1989) Parasitology Today, 5, pp. 111-116 
504 |a Pierres, Kourilsky, Rebonah, Dosseto, Gibbol, Distinct epitopes on Iak gene products identified by monoclonal antibodies (1980) European Journal of Immunology, 10, pp. 950-957 
504 |a Said, Joskowicz, Barreira, Eisen, Neuropathy associated with experimental Chagas disease (1985) Annals of Neurology, 18, pp. 676-683 
504 |a Wright, Hauptfeld, Lacy, Induction of Ia antigens expression on murine islet parenchymal cells does not dininish islet allograft survival (1989) American Journal of Pathology, 134, pp. 237-242 
520 3 |a In isolated skeletal, heart, and smooth muscle cells from BALB/c and C3H/HeJ mice infected with different strains of Trypanosoma cruzi the presence of class II MHC molecules was investigated by immunocytochemical techniques. We employed single muscle fibers instead of conventional cryostat sections to obtain a more accurate antigen localization. Approximately half of the skeletal muscle cells isolated from the reclus femoris expressed Ia antigens on their surface, irrespective of the mouse or parasite strain combination. Ia expression was apparent only at 30 days postinfection and thereafter. The heart muscle cells expressed class II molecules only at 1 and 3 months postinfection. In no case did the smooth muscle cells from infected mice express Ia antigens. Studies of the same molecules in the noninfected animals gave constantly negative results. We conclude that in the course of the chronic infection of mice with T. cruzi, ectopic expression of class II MHC molecules occurs at the surface of skeletal and heart muscle cells, providing a possible mechanism for explaining the anti-striped muscle autoreactivity present in Chagas' disease. © 1991.  |l eng 
536 |a Detalles de la financiación: Institut National de la Santé et de la Recherche Médicale 
536 |a Detalles de la financiación: Consejo Nacional de Investigaciones Científicas y Técnicas 
536 |a Detalles de la financiación: ACKNOWLEDGMENTS This work was partially supportedb y a collaborative grant from INSERM (France)-CONICET (Argentine). 
593 |a Cátedra de Patología II, Facultad de Ciencias Médicas, Universidad de La Plata, Calle 60 y 120, 1900 La Plata, Argentina 
593 |a Unité d' Immunoparasitologie, Institut Pasteur, 25, Rue du Dr. Roux, 75724 Paris Cedex 15, France 
593 |a Laboratorio de Biología Molecular de la Enfermedad de Chagas, I.N.G.E.B.I., Vuelta de Obligado 2490, 1428 Buenos Aires, Argentina 
690 1 0 |a AUTOIMMUNITY 
690 1 0 |a CHAGAS' DISEASE 
690 1 0 |a HEART MUSCLE CELLS 
690 1 0 |a IA ANTIGENS 
690 1 0 |a SKELETAL MUSCLE CELLS 
690 1 0 |a TRYPANOSOMA CRUZI 
690 1 0 |a ANTIGEN 
690 1 0 |a ANIMAL EXPERIMENT 
690 1 0 |a ANTIGEN EXPRESSION 
690 1 0 |a ARTICLE 
690 1 0 |a FEMALE 
690 1 0 |a MAJOR HISTOCOMPATIBILITY COMPLEX 
690 1 0 |a MOUSE 
690 1 0 |a NONHUMAN 
690 1 0 |a TRYPANOSOMA CRUZI 
690 1 0 |a ANIMAL 
690 1 0 |a CELLS, CULTURED 
690 1 0 |a CHAGAS DISEASE 
690 1 0 |a FEMALE 
690 1 0 |a HEART 
690 1 0 |a HISTOCOMPATIBILITY ANTIGENS CLASS II 
690 1 0 |a IMMUNOHISTOCHEMISTRY 
690 1 0 |a MALE 
690 1 0 |a MICE 
690 1 0 |a MICE, INBRED BALB C 
690 1 0 |a MICE, INBRED C3H 
690 1 0 |a MUSCLE, SMOOTH 
690 1 0 |a MUSCLES 
690 1 0 |a SUPPORT, NON-U.S. GOV'T 
690 1 0 |a TRYPANOSOMA CRUZI 
690 1 0 |a ANIMALIA 
690 1 0 |a TRYPANOSOMA 
690 1 0 |a TRYPANOSOMA CRUZI 
700 1 |a Hontebeyrie-Joskowicz, M. 
700 1 |a Chambo, J. 
700 1 |a Levin, M. 
700 1 |a Laguens, R.P. 
773 0 |d 1991  |g v. 72  |h pp. 8-14  |k n. 1  |p Exp. Parasitol.  |x 00144894  |w (AR-BaUEN)CENRE-4717  |t Experimental Parasitology 
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856 4 0 |u https://hdl.handle.net/20.500.12110/paper_00144894_v72_n1_p8_Meckert  |y Handle 
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