Activation of GABA B receptors in the anterior pituitary inhibits prolactin and luteinizing hormone secretion

Previous work from our laboratory showed that baclofen could lower serum prolactin (PRL) levels acting at the central nervous system. The present experiments were designed to evaluate whether the γ-aminobutyric acid B agonist was also effective in inhibiting hormone release at the pituitary level. I...

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Autor principal: Lux-Lantos, V.
Otros Autores: Rey, E., Libertum, C.
Formato: Capítulo de libro
Lenguaje:Inglés
Publicado: 1992
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Acceso en línea:Registro en Scopus
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Registro en la Biblioteca Digital
Aporte de:Registro referencial: Solicitar el recurso aquí
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008 190411s1992 xx ||||fo|||| 00| 0 eng|d
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024 7 |2 cas  |a 3 aminopropanephosphonic acid, 13138-33-5; baclofen, 1134-47-0; luteinizing hormone, 39341-83-8, 9002-67-9; phaclofen, 108351-35-5; prolactin, 12585-34-1, 50647-00-2, 9002-62-4; protirelin, 24305-27-9 
040 |a Scopus  |b spa  |c AR-BaUEN  |d AR-BaUEN 
030 |a NUNDA 
100 1 |a Lux-Lantos, V. 
245 1 0 |a Activation of GABA B receptors in the anterior pituitary inhibits prolactin and luteinizing hormone secretion 
260 |c 1992 
270 1 0 |m Libertum, C.; Laboratorio de Neuroendocrinologia, Inst de Biologia y Medicina Exptl, V. de Obligado 2490, 1428 Buenos Aires, Argentina 
506 |2 openaire  |e Política editorial 
520 3 |a Previous work from our laboratory showed that baclofen could lower serum prolactin (PRL) levels acting at the central nervous system. The present experiments were designed to evaluate whether the γ-aminobutyric acid B agonist was also effective in inhibiting hormone release at the pituitary level. In monolayer cultures of adenohypophyseal dispersed cells, baclofen inhibited basal PRL secetion after 1 or 2 h of incubation. This inhibition was significantly abolished by three antagonists: phaclofen, 3-aminopropyl-phosphonic acid and 4-aminobutylphosphonic acid. Furthermore, baclofen inhibited the thyrotropin-releasing hormone-induced PRL release in a concentration-dependent manner. With regard to gonadotropin secretion, baclofen was unable to modify basal luteinizing hormone (LH) secretion, but significantly inhibited the LH-releasing hormone-induced LH release. These results show that baclofen, in addition to its central neuroendocrine effects, inhibits pituitary hormone secretion, under basal and/or stimulated conditions, by direct action at the pituitary level.  |l eng 
593 |a Laboratorio de Neuroendocrinologia, Inst de Biologia y Medicina Exptl, V. de Obligado 2490, 1428 Buenos Aires, Argentina 
690 1 0 |a ADENOHYPOPHYSEAL CELL CULTURES 
690 1 0 |a BACLOFEN 
690 1 0 |a LUTEINIZING HORMONE 
690 1 0 |a PROLACTIN 
690 1 0 |a 3 AMINOPROPANEPHOSPHONIC ACID 
690 1 0 |a 4 AMINOBUTANEPHOSPHONIC ACID 
690 1 0 |a 4 AMINOBUTYRIC ACID B RECEPTOR 
690 1 0 |a BACLOFEN 
690 1 0 |a LUTEINIZING HORMONE 
690 1 0 |a PHACLOFEN 
690 1 0 |a PROLACTIN 
690 1 0 |a PROTIRELIN 
690 1 0 |a ADENOHYPOPHYSIS 
690 1 0 |a ANIMAL CELL 
690 1 0 |a ANIMAL EXPERIMENT 
690 1 0 |a ARTICLE 
690 1 0 |a CONTROLLED STUDY 
690 1 0 |a FEMALE 
690 1 0 |a GONADOTROPIN RELEASE 
690 1 0 |a HYPOPHYSIS CELL 
690 1 0 |a LUTEINIZING HORMONE RELEASE 
690 1 0 |a MONOLAYER CULTURE 
690 1 0 |a NONHUMAN 
690 1 0 |a PRIORITY JOURNAL 
690 1 0 |a PROLACTIN RELEASE 
690 1 0 |a RAT 
653 0 0 |a lioresal, Ciba Geigy, Argentina 
700 1 |a Rey, E. 
700 1 |a Libertum, C. 
773 0 |d 1992  |g v. 56  |h pp. 687-693  |k n. 5  |p NEUROENDOCRINOLOGY  |x 00283835  |w (AR-BaUEN)CENRE-6253  |t Neuroendocrinology 
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856 4 0 |u https://doi.org/10.1159/000126294  |y DOI 
856 4 0 |u https://hdl.handle.net/20.500.12110/paper_00283835_v56_n5_p687_LuxLantos  |y Handle 
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