Peripheral ocular motor disorders

In the reviewed period, articles on peripheral eye movement disorders covered interesting aspects. Localizing value of associated signs, repetitive presentations of palsies, and classical quotations are stressed for the oculomotor nerve. The superior oblique is correlated to central nervous system d...

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Detalles Bibliográficos
Autor principal: Ebner, R.
Formato: Capítulo de libro
Lenguaje:Inglés
Publicado: 1994
Materias:
Acceso en línea:Registro en Scopus
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Registro en la Biblioteca Digital
Aporte de:Registro referencial: Solicitar el recurso aquí
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024 7 |2 cas  |a DNA, Mitochondrial 
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100 1 |a Ebner, R. 
245 1 0 |a Peripheral ocular motor disorders 
260 |c 1994 
270 1 0 |m Ebner, R.Cbel. Diaz 2277/5 'D', Buenos Aires 1428, Argentina 
506 |2 openaire  |e Política editorial 
520 3 |a In the reviewed period, articles on peripheral eye movement disorders covered interesting aspects. Localizing value of associated signs, repetitive presentations of palsies, and classical quotations are stressed for the oculomotor nerve. The superior oblique is correlated to central nervous system disorders when overacting in pediatric patients or when ocular torsion is matched to the perceived vertical tilt. The family 'pseudo' brought two of its members: 'pseudo' myasthenia and 'pseudo' myotonia. Mitochondrial citopathies with ocular manifestations can overlap among the different clinical types, eg, Kearns-Sayre, MELAS (mitochondrial encephalopathy-lactic acidosis and strokelike episodes), MERFF (myoclonic epilepsy and ragged red fibers). The diagnostic value of DNA mutations is emphasized in those syndromes. Imaging of the carotid arteries provides useful hints in cases where the lumen is narrowed due to internal processes or external compression; its interpretation is not only of diagnostic but of prognostic value. Certain otorhinolaryngology surgical procedures can damage the orbital muscles and produce serious inconvenience to the ocular motility. Analyzing the involved structures the therapeutic gesture can be determined. Diplopia after cataract surgery or retinal detachment repair is due to different factors, anesthetics, or implant location and is implied in every case.  |l eng 
593 |a Cbel. Diaz 2277/5 'D', Buenos Aires 1428, Argentina 
690 1 0 |a MITOCHONDRIAL DNA 
690 1 0 |a ABDUCENS NERVE PARALYSIS 
690 1 0 |a ARTERIOGRAPHY 
690 1 0 |a BRAIN INJURY 
690 1 0 |a CAROTID ARTERY OBSTRUCTION 
690 1 0 |a CLINICAL FEATURE 
690 1 0 |a CRANIAL NERVE PARALYSIS 
690 1 0 |a DIGITAL SUBTRACTION ANGIOGRAPHY 
690 1 0 |a DIPLOPIA 
690 1 0 |a DURA MATER 
690 1 0 |a EYE MOVEMENT DISORDER 
690 1 0 |a EYE SURGERY 
690 1 0 |a HUMAN 
690 1 0 |a MAGNETIC RESONANCE ANGIOGRAPHY 
690 1 0 |a MITOCHONDRIAL MYOPATHY 
690 1 0 |a MYASTHENIA 
690 1 0 |a OPHTHALMOPLEGIA 
690 1 0 |a POINT MUTATION 
690 1 0 |a PRIORITY JOURNAL 
690 1 0 |a PTOSIS 
690 1 0 |a RETINA DETACHMENT 
690 1 0 |a REVIEW 
690 1 0 |a SCLERA BUCKLING PROCEDURE 
690 1 0 |a SUPERIOR OBLIQUE MUSCLE 
690 1 0 |a TROCHLEAR NERVE 
690 1 0 |a VITRECTOMY 
690 1 0 |a CAROTID ARTERY DISEASES 
690 1 0 |a DIAGNOSIS, DIFFERENTIAL 
690 1 0 |a DNA, MITOCHONDRIAL 
690 1 0 |a HUMAN 
690 1 0 |a MITOCHONDRIAL MYOPATHIES 
690 1 0 |a MYASTHENIA GRAVIS 
690 1 0 |a MYOTONIA 
690 1 0 |a OCULAR MOTILITY DISORDERS 
690 1 0 |a OCULOMOTOR MUSCLES 
690 1 0 |a OCULOMOTOR NERVE 
690 1 0 |a POINT MUTATION 
650 1 7 |2 spines  |a MENINGITIS 
650 1 7 |2 spines  |a TORSION 
773 0 |d 1994  |g v. 5  |h pp. 23-28  |k n. 6  |p CURR. OPIN. OPHTHALMOL.  |x 10408738  |t Current Opinion in Ophthalmology 
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