Glucocorticoid negative feedback and glucocorticoid receptors after hippocampectomy in rats

In rats with dorsal hippocampectomy, glucocorticoid receptors in the hypothalamus and anterior pituitary, as well as the pituitary transcortin-like compound, are preserved, in spite of a 60% depletion of glucocorticoid receptors in the hippocampus. In the hippocampectomized group, basal levels of se...

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Detalles Bibliográficos
Autor principal: Magarinos, A.M
Otros Autores: Somoza, G., De Nicola, A.F
Formato: Capítulo de libro
Lenguaje:Inglés
Publicado: 1987
Acceso en línea:Registro en Scopus
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Registro en la Biblioteca Digital
Aporte de:Registro referencial: Solicitar el recurso aquí
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100 1 |a Magarinos, A.M. 
245 1 0 |a Glucocorticoid negative feedback and glucocorticoid receptors after hippocampectomy in rats 
260 |c 1987 
506 |2 openaire  |e Política editorial 
520 3 |a In rats with dorsal hippocampectomy, glucocorticoid receptors in the hypothalamus and anterior pituitary, as well as the pituitary transcortin-like compound, are preserved, in spite of a 60% depletion of glucocorticoid receptors in the hippocampus. In the hippocampectomized group, basal levels of serum corticosterone (CORT) were increased, although there was a normal response to ether stress. Inhibition of the response to ether with dexamethasone (DEX) was dose-dependent: whereas 100 μg/kg completely suppressed serum CORT, 10 μg/kg were ineffective. However, we observed a reduced sensitivity to DEX inhibition with 25 μg/kg in hippocampectomized animals. These results indicate that the hippocampus is involved in negative feedback mechanisms, although different doses of DEX are needed for this demonstration. The inhibition of serum CORT due to 100 μg/kg DEX suggests that negative feedback at sites other than the hippocampus was still operative, in agreement with normal levels of glucocorticoid receptors in the anterior pituitary and hypothalamus of hippocampectomized rats.  |l eng 
593 |a Laboratorios de Esteroides and Neuroendocrinologia, Instituto de Biologia y Medicina Experimental, 1428 Buenos Aires, Argentina 
690 1 0 |a DEXAMETHASONE 
690 1 0 |a GLUCOCORTICOID 
690 1 0 |a ADENOHYPOPHYSIS 
690 1 0 |a ANIMAL EXPERIMENT 
690 1 0 |a BIOLOGICAL MODEL 
690 1 0 |a ENDOCRINE SYSTEM 
690 1 0 |a FEEDBACK SYSTEM 
690 1 0 |a HIPPOCAMPUS 
690 1 0 |a NONHUMAN 
690 1 0 |a PRIORITY JOURNAL 
690 1 0 |a RAT 
690 1 0 |a STRESS 
690 1 0 |a ANIMALIA 
700 1 |a Somoza, G. 
700 1 |a De Nicola, A.F. 
773 0 |d 1987  |g v. 19  |h pp. 105-109  |k n. 3  |p HORM. METAB. RES.  |x 00185043  |t Hormone and Metabolic Research 
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