Effect of hexavalent chromium on proliferation and differentiation to adipocytes of 3T3-L1 fibroblasts

Heavy metals contamination has become an important risk factor for public health and the environment. Chromium is a frequent industrial contaminant and is also used in orthopaedic joint replacements made from cobalt-chromium-alloy. Since hexavalent chromium (Cr(VI)) was reported as genotoxic and car...

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Autor principal: Martini, C.N
Otros Autores: Brandani, J.N, Gabrielli, M., Vila, M.D.C
Formato: Capítulo de libro
Lenguaje:Inglés
Publicado: Elsevier Ltd 2014
Acceso en línea:Registro en Scopus
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024 7 |2 scopus  |a 2-s2.0-84896141428 
024 7 |2 cas  |a chromium, 16065-83-1, 7440-47-3, 14092-98-9; dexamethasone, 50-02-2; insulin, 9004-10-8; isobutylmethylxanthine, 28822-58-4; Chromium; chromium hexavalent ion; Environmental Pollutants 
040 |a Scopus  |b spa  |c AR-BaUEN  |d AR-BaUEN 
030 |a TIVIE 
100 1 |a Martini, C.N. 
245 1 0 |a Effect of hexavalent chromium on proliferation and differentiation to adipocytes of 3T3-L1 fibroblasts 
260 |b Elsevier Ltd  |c 2014 
270 1 0 |m Vila, M.D.C.; Departamento de Química Biológica, Facultad de Ciencias Exactas y Naturales, Universidad de Buenos Aires, Pabellón 2, Ciudad Universitaria, 1428 Buenos Aires, Argentina; email: mvila@qb.fcen.uba.ar 
506 |2 openaire  |e Política editorial 
504 |a Bradford, M., A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye-binding (1976) Anal. Biochem., 72, pp. 248-254 
504 |a Davies, A.P., Sood, A., Lewis, A.C., Newson, R., Learmonth, I.D., Case, C.P., Metal-specific differences in levels of DNA damage caused by synovial fluid recovered at revision arthroplasty (2005) J. Bone Joint Surg. Br., 87, pp. 1439-1444 
504 |a Farmer, S.R., Transcriptional control of adipocyte formation (2006) Cell. Metab., 4, pp. 263-273 
504 |a Figgitt, M., Newson, R., Leslie, I.J., Fisher, J., Ingham, E., Case, C.P., The genotoxicity of physiological concentrations of chromium (Cr(III) and Cr(VI)) and cobalt (Co(II)): an in vitro study (2010) Mutat. Res., 688, pp. 53-61 
504 |a Kawanishi, S., Hiraku, Y., Murata, M., Oikawa, S., The role of metals in sitespecific DNA damage with reference to carcinogenesis (2002) Free Radic. Biol. Med., 32, pp. 822-832 
504 |a Keegan, G.M., Learmonth, I.D., Case, C.P., A systematic comparison of the actual, potential, and theoretical health effects of cobalt and chromium exposures from industry and surgical implants (2008) Crit. Rev. Toxicol., 38, pp. 645-674 
504 |a Kopec, A.K., Thompson, C.M., Kim, S., Forgacs, A.L., Zacharewski, T.R., Comparative toxicogenomic analysis of oral Cr(VI) exposure effects in rat and mouse small intestinal epithelia (2012) Toxicol. Appl. Pharmacol., 262, pp. 124-138 
504 |a Laemmli, U.K., Cleavage of structural proteins during the assembly of the head of bacteriophage T4 (1970) Nature, 227, pp. 680-685 
504 |a Lukas, J., Bartkova, J., Rohde, M., Strauss, M., Bartek, J., Cyclin D1 is dispensable for G1 control in retinoblastoma gene-deficient cells independently of cdk4 activity (1995) Mol. Cell. Biol., 15, pp. 2600-2611 
504 |a Marouani, N., Tebourbi, O., Mahjoub, S., Yacoubi, M.T., Sakly, M., Benkhalifa, M., Rhouma, K.B., Effects of hexavalent chromiun on reproductive functions of male adult rats (2012) Reprod. Biol., 12, pp. 119-133 
504 |a Martini, C., Plaza, M.V., Vila, M.C., PKA-dependent and independent cAMP signalling in 3T3-L1 fibroblasts differentiation (2009) Mol. Cell. Endocrinol., 298, pp. 42-47 
504 |a Martini, C.N., Gabrielli, M., Vila, M.C., A commercial formulation of glyphosate inhibits proliferation and differentiation to adipocytes and induces apoptosis in 3T3-L1 fibroblasts (2012) Toxicol. In Vitro, 26, pp. 1007-1013 
504 |a Qiu, Z., Wei, Y., Chen, N., Jiang, M., Wu, J., Liao, K., DNA synthesis and mitotic clonal expansion is not a required step for 3T3-L1 preadipocytes into adipocytes (2001) J. Biol. Chem., 276, pp. 11988-11995 
504 |a Raghunathan, V.K., Ellis, E.M., Grant, M.H., Response to chronic exposure to hexavalent chromium in human monocytes (2009) Toxicol. In Vitro, 23, pp. 647-652 
504 |a Salnikow, K., Zhitkovich, A., Genetic and epigenetic mechanisms in metal carcinogenesis and cocarcinogenesis: nickel, arsenic, and chromium (2008) Chem. Res. Toxicol., 21, pp. 28-44 
504 |a Samuel, J.B., Stanley, J.A., Vengatesh, G., Princess, R.A., Muthusami, S., Roopha, D.P., Suthagar, E., Aruldhas, M.M., Ameliorative effect of vitamin C on hexavalent chromium-induced delay in sexual maturation and oxidative stress in developing Wistar rat ovary and uterus (2012) Toxicol. Ind. Health, 28, pp. 720-733 
504 |a Wang, Z.X., Jiang, C.S., Liu, L., Wang, X.H., Jin, H.J., Wu, Q., Chen, Q., The role of AKT on arsenic trioxide suppression of 3T3-L1 preadipocyte differentiation (2005) Cell Res., 15, pp. 379-386 
504 |a Witt, K.L., Stout, M.D., Herbert, R.A., Travlos, G.S., Kissling, G.E., Collins, B.J., Hooth, M.J., Mechanistic insights from de NTP studies of chromium (2013) Toxicol. Pathol., 41, pp. 326-342 
520 3 |a Heavy metals contamination has become an important risk factor for public health and the environment. Chromium is a frequent industrial contaminant and is also used in orthopaedic joint replacements made from cobalt-chromium-alloy. Since hexavalent chromium (Cr(VI)) was reported as genotoxic and carcinogenic in different mammals, to further evaluate its cytotoxicity, we investigated the effect of this heavy metal in the proliferation and differentiation to adipocytes of 3T3-L1 fibroblasts. These cells, after the addition of a mixture containing insulin, dexamethasone and methylisobutylxanthine, first proliferate, a process known as mitotic clonal expansion (MCE), and then differentiate to adipocytes. In this differentiation process a key transcription factor is induced: peroxisome proliferator-activated receptor gamma (PPAR gamma). We found that treatment of 3T3-L1 fibroblasts with potassium chromate inhibited proliferation in exponentially growing cells and MCE as well as differentiation. A decrease in PPAR gamma content, evaluated by western blot and immunofluorescence, was found in cells differentiated in the presence of chromium. On the other hand, after inhibition of differentiation with chromium, when the metal was removed, differentiation was recovered, which indicates that this may be a reversible effect. We also found an increase in the number of micronucleated cells after treatment with Cr(VI) which is associated with genotoxic effects. According to our results, Cr(VI) is able to inhibit proliferation and differentiation to adipocytes of 3T3-L1 fibroblasts and to increase micronucleated cells, which are all indicative of alterations in cellular physiology and therefore, contributes to further elucidate the cytotoxic effects of this heavy metal. © 2014 Elsevier Ltd.  |l eng 
536 |a Detalles de la financiación: Agencia Nacional de Promoción Científica y Tecnológica 
536 |a Detalles de la financiación: Consejo Nacional de Investigaciones Científicas y Técnicas 
536 |a Detalles de la financiación: This work was supported by research grants from Agencia Nacional de Promoción Científica y Tecnológica and Consejo Nacional de Investigaciones Científicas y Técnicas of Argentina. The funders had no role in study design, data and analysis, decision to publish, or preparation of the manuscript. 
593 |a Departamento de Química Biológica, Facultad de Ciencias Exactas y Naturales, Universidad de Buenos Aires, Pabellón 2, Ciudad Universitaria, 1428 Buenos Aires, Argentina 
690 1 0 |a 3T3-L1 FIBROBLASTS 
690 1 0 |a ADIPOGENESIS 
690 1 0 |a HEXAVALENT CHROMIUM 
690 1 0 |a PPAR GAMMA 
690 1 0 |a PROLIFERATION 
690 1 0 |a CHROMIUM 
690 1 0 |a DEXAMETHASONE 
690 1 0 |a INSULIN 
690 1 0 |a ISOBUTYLMETHYLXANTHINE 
690 1 0 |a PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA 
690 1 0 |a TRIACYLGLYCEROL 
690 1 0 |a CHROMIUM 
690 1 0 |a CHROMIUM HEXAVALENT ION 
690 1 0 |a POLLUTANT 
690 1 0 |a ADIPOCYTE 
690 1 0 |a ANIMAL CELL 
690 1 0 |a ARTICLE 
690 1 0 |a CELL COUNT 
690 1 0 |a CELL DIFFERENTIATION 
690 1 0 |a CELL EXPANSION 
690 1 0 |a CELL PROLIFERATION 
690 1 0 |a CELL STRAIN 3T3 
690 1 0 |a COMPARATIVE STUDY 
690 1 0 |a CONTROLLED STUDY 
690 1 0 |a CYTOTOXICITY TEST 
690 1 0 |a EMBRYO 
690 1 0 |a IMMUNOFLUORESCENCE 
690 1 0 |a LIPID STORAGE 
690 1 0 |a MICRONUCLEUS 
690 1 0 |a MITOTIC CLONAL EXPANSION 
690 1 0 |a MOLECULAR DYNAMICS 
690 1 0 |a MOUSE 
690 1 0 |a NONHUMAN 
690 1 0 |a WESTERN BLOTTING 
690 1 0 |a 3T3 CELL LINE 
690 1 0 |a ADIPOCYTE 
690 1 0 |a ANIMAL 
690 1 0 |a CELL DIFFERENTIATION 
690 1 0 |a CELL PROLIFERATION 
690 1 0 |a CYTOLOGY 
690 1 0 |a DRUG EFFECTS 
690 1 0 |a FIBROBLAST 
690 1 0 |a PHYSIOLOGY 
690 1 0 |a POLLUTANT 
690 1 0 |a TOXICITY 
690 1 0 |a 3T3-L1 CELLS 
690 1 0 |a ADIPOCYTES 
690 1 0 |a ANIMALS 
690 1 0 |a CELL DIFFERENTIATION 
690 1 0 |a CELL PROLIFERATION 
690 1 0 |a CHROMIUM 
690 1 0 |a ENVIRONMENTAL POLLUTANTS 
690 1 0 |a FIBROBLASTS 
690 1 0 |a MICE 
700 1 |a Brandani, J.N. 
700 1 |a Gabrielli, M. 
700 1 |a Vila, M.D.C. 
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