Porphyrinogenesis in rat cerebellum. Effect of high δ-aminolevulinic acid concentration

1. δ-aminolevulinic acid (ALA) uptake as well as precursor accumulation and porphyrin biosynthesis were investigated in rat cerebellum, using as experimental approach minimal tissue units called particles. 2. ALA was shown to be taken up into cerebellum particles by a non saturable process up to 4.0...

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Autor principal: Princ, F.G
Otros Autores: Juknat, A.A, Del Carmen Batlle, A.M
Formato: Capítulo de libro
Lenguaje:Inglés
Publicado: 1994
Acceso en línea:Registro en Scopus
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Registro en la Biblioteca Digital
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024 7 |2 scopus  |a 2-s2.0-0028235334 
024 7 |2 cas  |a Aminolevulinic Acid, 106-60-5; Glucose, 50-99-7; Porphyrins 
040 |a Scopus  |b spa  |c AR-BaUEN  |d AR-BaUEN 
030 |a GEPHD 
100 1 |a Princ, F.G. 
245 1 0 |a Porphyrinogenesis in rat cerebellum. Effect of high δ-aminolevulinic acid concentration 
260 |c 1994 
270 1 0 |m Del Carmen Batlle, A.M. 
506 |2 openaire  |e Política editorial 
504 |a Alperin, Idoyaga-Vargas, Carminatti, Rate of protein glycosylation in rat cerebral cortex (1986) J. Neurochem., 47, pp. 355-362 
504 |a Becker, Viljoen, Kramer, The inhibition of red cell and brain ATPase by δ-aminolevulinic acid (1971) Biochim. biophys. Acta, 225, pp. 26-34 
504 |a Becker, Kramer, Viljoen, Delta-aminolevulinic acid uptake by rabbit brain cerebral cortex (1974) J. Neurochem., 23, pp. 1019-1023 
504 |a Becker, Viljoen, Kramer, Porphyrin precursors and their effects in vitro on some aspects of nerve function (1976) Porphyrins in human disease. First International Porphyrin Meeting, pp. 163-172. , M. Doss, Karger, Basel 
504 |a Bonkowsky, Schady, Neurologic manifestations of acute porphyria (1982) Semin. Liver Dis., 2, pp. 108-124 
504 |a Brennan, Cantrill, δ-Aminolevulinic acid is a potent agonist for GABA autoreceptors (1979) Nature, 280, pp. 514-515 
504 |a Cutler, Moore, Ewart, Effects of delta-aminolevulinic acid administration on social behaviour in the laboratory mouse (1979) Psychopharmacology, 61, pp. 131-135 
504 |a De Matteis, Ray, Studies on cerebellar haem metabolism in the rat in vivo (1982) J. Neurochem., 39, pp. 551-556 
504 |a Durko, Juhasz, In vitro distribution of porphyrin metabolites from 10−3 M delta-aminolevulinic acid in primary neural tissue cultures (1987) Neurochemical Research, 12, pp. 465-468 
504 |a Elder, Acquired disorders of haem synthesis (1976) Essays Med. Biochem., 2, pp. 75-114 
504 |a Hermes-Lima, Valle, Vercesi, Bechara, Damage to rat liver mitochondria promoted by δ-aminolevulinic acid-generated reactive oxygen species: connections with acute intermittent porphyria and lead-poisoning (1991) Biochim. Biophys. Acta, 1056, pp. 57-63 
504 |a Hermes-Lima, Castilho, Valle, Bechara, Vercesi, Calcium-dependent mitochondrial oxidative damage promoted by 5-aminolevulinic acid (1992) Biochim. biophys. Acta, 1180, pp. 201-206 
504 |a Kotler, Juknat, Correa Garcia, Batlle, The action of sulphamerazine on porphyrin biosynthesis in rats (1988) Med. Sci. Res., 16, pp. 323-324 
504 |a Lowry, Rosebrough, Farr, Randall, Protein measurement with the folin phenol reagent (1951) J. biol. Chem., 193, pp. 265-275 
504 |a Maines, Regional distribution of the enzymes of haem biosynthesis and the inhibition of 5-aminolevulinate synthase by manganese in the rat brain (1980) Biochem. J., 190, pp. 315-321 
504 |a Mauzerall, Granick, The occurrence and determination of δ-aminolevulinic acid and porphobilinogen in urine (1956) J. biol. Chem., 219, pp. 435-446 
504 |a Percy, Shanley, Studies on haem biosynthesis in rat brain (1979) J. Neurochem., 33, pp. 1267-1274 
504 |a Percy, Lamm, Taljaard, δ-aminolevulinic acid uptake, toxicity and effect on [14C]aminobutiric acid uptake into neurons and glia in culture (1981) J. Neurochem., 36, pp. 69-76 
504 |a Russell, Lamm, Taljaard, Effects of δ-aminolevulinic acid, porphobilinogen and structurally related amino acids on 2-deoxy-glucose uptake in cultured neurons (1982) Neurochem. Res., 7, pp. 1009-1022 
504 |a Russell, Lamm, Taljaard, Inhibition of Na+, K+-ATPase activity by δ-aminolevulinic acid (1983) Neurochem. Res., 8, pp. 1407-1415 
504 |a Sima, Kennedy, Blakeslee, Robertson, Experimental porphyric neuropathy: a preliminary report (1981) Can. J. Neurol. Sci., 8, pp. 105-114 
504 |a Sokoloff, Relation between physiological function and energy metabolism in the central nervous system (1977) J. Neurochem., 29, pp. 13-26 
504 |a Sweeney, Pathak, Asbury, Acute intermittent porphyria (1970) Increased ALA-synthetase activity during an acute attack, 93, pp. 369-380. , Brain 
504 |a Trinder, Colorimetric method for glucose determination in blood and other biological fluids (1969) Annals of Clinical Biochemistry: An international journal of biochemistry and laboratory medicine, 6, p. 24 
504 |a Wetterberg, Report on an international survey of safe and unsafe drugs in acute and intermittent porphyria (1976) Porphyrins in Human Diseases—Report of Discussions, pp. 191-202. , M. Doss, P. Nawrocki, Falk, Freiburg 
504 |a Whetsell, Sassa, Bickers, Kappas, Studies on porphyrin-heme biosynthesis in organotypic cultures of chick dorsal root ganglion (1978) J. Neuropath. exp. Neurol., 37, pp. 497-507 
504 |a Yeung Laiwah, Goldberg, Moore, Pathogenesis and treatment of acute intermittent porphyria: discussion paper (1983) J. R. Soc. Med., 76, pp. 386-392 
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520 3 |a 1. δ-aminolevulinic acid (ALA) uptake as well as precursor accumulation and porphyrin biosynthesis were investigated in rat cerebellum, using as experimental approach minimal tissue units called particles. 2. ALA was shown to be taken up into cerebellum particles by a non saturable process up to 4.0 mM ALA whereas PBG and porphyrin formation exhibited a hyperbolic response reaching the plateau at about 1.0 and 1.5 mM ALA respectively. 3. Exposure of cerebellum particles to high exogenous ALA amounts (0.01-4.0 mM) indicated that ALA can be accumulated in relatively high concentrations in the cells (40 nmol/mg protein). Under these experimental conditions, PBG-D presented a low activity (3.25 pmol/mg protein/4 hr) showing to be a secondary control step in heme biosynthesis. 4. Incubation of cerebellum particles in the presence of a physiological concentration of glucose revealed that 1.0 mM ALA decreased glucose uptake by the cells (87% during 1 hr incubation), being consistent with the fact that acute attacks are precipitated by fasting and that sugar administration appeared to be an efficient treatment of AIP crisis. 5. These findings provide the basis for a useful model to study the nature of the metabolic mechanism underlying the acute attack. © 1994.  |l eng 
536 |a Detalles de la financiación: Alexander von Humboldt-Stiftung, 8151/90011 
536 |a Detalles de la financiación: National Council for Scientific Research 
536 |a Detalles de la financiación: Consejo Nacional de Investigaciones Científicas y Técnicas 
536 |a Detalles de la financiación: Acknowledgements--A. M. del C. Batlle and A. A, Juknat hold the posts of Superior and Associate Scientific Researchers in the Argentine National Research Council (CONICET). F. G. Princ is a Research Assistant at the CONICET. This work forms part of the Thesis of F. G. Princ to be submitted to the University of Buenos Aires for his Ph.D. degree. The support of CONICET and CEDIQUIFA is gratefully acknowledged. A. A. Juknat is indebted to the Alexander von Humboldt Foundation (Bonn, Germany) for providing a grant (8151/90011). The authors also wish to thank Dr Victor Idoyaga Vargas for his most stimulating discussion, and Dr Susana Afonso for the excellent drawings. 
593 |a Centro de Investigaciones sobre Porfirinas y Porfirias (CIPYP), (CONICET), Facultad de Ciencias Exactas y Naturales, Universidad de Buenos Aires, Viamonte 1881 10A 1056 CP-Buenos Aires, Argentina [Fax: 54 1 811 7447] 
690 1 0 |a PORPHYRIA 
690 1 0 |a PORPHYRINOGENESIS 
690 1 0 |a RAT CEREBELLUM 
690 1 0 |a Δ-AMINOLEVULINIC ACID 
690 1 0 |a AMINOLEVULINIC ACID 
690 1 0 |a GLUCOSE 
690 1 0 |a PORPHOBILINOGEN 
690 1 0 |a PORPHOBILINOGEN DEAMINASE 
690 1 0 |a PORPHYRIN 
690 1 0 |a ANIMAL TISSUE 
690 1 0 |a ARTICLE 
690 1 0 |a CEREBELLUM 
690 1 0 |a CONTROLLED STUDY 
690 1 0 |a DRUG ACCUMULATION 
690 1 0 |a GLUCOSE TRANSPORT 
690 1 0 |a MALE 
690 1 0 |a NONHUMAN 
690 1 0 |a PORPHYRIA 
690 1 0 |a PORPHYRIN METABOLISM 
690 1 0 |a PRIORITY JOURNAL 
690 1 0 |a RAT 
690 1 0 |a AMINOLEVULINIC ACID 
690 1 0 |a ANIMAL 
690 1 0 |a CEREBELLUM 
690 1 0 |a GLUCOSE 
690 1 0 |a MALE 
690 1 0 |a PORPHYRINS 
690 1 0 |a RATS 
690 1 0 |a SUPPORT, NON-U.S. GOV'T 
700 1 |a Juknat, A.A. 
700 1 |a Del Carmen Batlle, A.M. 
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