Galectin-1 Prevents Infection and Damage Induced by Trypanosoma cruzi on Cardiac Cells

Background: Chronic Chagas cardiomyopathy caused by Trypanosoma cruzi is the result of a pathologic process starting during the acute phase of parasite infection. Among different factors, the specific recognition of glycan structures by glycan-binding proteins from the parasite or from the mammalian...

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Detalles Bibliográficos
Autores principales: Benatar, A.F., García, G.A., Bua, J., Cerliani, J.P., Postan, M., Tasso, L.M., Scaglione, J., Stupirski, J.C., Toscano, M.A., Rabinovich, G.A., Gómez, K.A.
Formato: JOUR
Materias:
agglutinin
galectin 1
lipocortin 5
phytohemagglutinin
adult
aged
animal cell
Article
cell invasion assay
Chagas disease
clinical article
clinical assessment
controlled study
Doppler echocardiography
electrocardiography
enzyme linked immunosorbent assay
flow cytometry
gene deletion
genetic transfection
glycosylation
heart transplantation
histopathology
host parasite interaction
human
image analysis
immunoblotting
mouse
nonhuman
protein expression
reverse transcription polymerase chain reaction
RNA extraction
survival
thorax radiography
Trypanosoma cruzi
trypomastigote
animal
Brazil
cardiac muscle cell
cell culture
disease model
female
immunology
knockout mouse
male
metabolism
middle aged
parasitemia
parasitology
pathology
physiology
survival analysis
Adult
Aged
Animals
Cells, Cultured
Chagas Disease
Disease Models, Animal
Female
Galectin 1
Host-Parasite Interactions
Humans
Male
Mice
Mice, Knockout
Middle Aged
Myocytes, Cardiac
Parasitemia
Survival Analysis
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_19352727_v9_n10_p_Benatar
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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