Calcium channels and synaptic transmission in familial hemiplegic migraine type 1 animal models

One of the outstanding developments in clinical neurology has been the identification of ion channel mutations as the origin of a wide variety of inherited disorders like migraine, epilepsy, and ataxia. The study of several channelopathies has provided crucial insights into the molecular mechanisms,...

Descripción completa

Guardado en:
Detalles Bibliográficos
Autores principales: Uchitel, O.D., González Inchauspe, C., Di Guilmi, M.N.
Formato: JOUR
Materias:
Cav2.1 (P/Q-Type) Ca2+ channels
Cortical spreading depression (CSD)
Familial hemiplegic migraine
R192Q and S218L knock in mice
Synaptic transmission
calcium channel 2.1
unclassified drug
voltage gated calcium channel
ataxia
CACNA1A gene mutation
circadian rhythm
familial hemiplegic migraine
familial hemiplegic migraine type 1
gene mutation
headache
human
migraine
mouse
nerve cell plasticity
nonhuman
priority journal
review
spreading cortical depression
synaptic transmission
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_18672450_v6_n1_p15_Uchitel
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

Ejemplares similares

Ejemplares similares