Tumor necrosis factor alpha induces LIF expression through ERK1/2 activation in mammary epithelial cells

It has been reported that expression of tumor necrosis factor superfamily members occur at the onset of the mammary gland post-lactational involution. One of these proteins, tumor necrosis factor alpha (TNFα), is a major mediator of inflammation that is able to induce expression of several cytokines...

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Detalles Bibliográficos
Autores principales: Levy, C.S., Slomiansky, V., Gattelli, A., Nahmod, K., Pelisch, F., Blaustein, M., Srebrow, A., Coso, O.A., Kordon, E.C.
Formato: JOUR
Materias:
AP-1
Involution
LIF
Mammary epithelial cells
Mammary gland
TNFα
leukemia inhibitory factor
mitogen activated protein kinase 1
mitogen activated protein kinase 3
transcription factor AP 1
tumor necrosis factor alpha
tumor necrosis factor alpha receptor
tumor necrosis factor alpha receptor 2
unclassified drug
LIF protein, human
MAPK1 protein, human
animal cell
animal experiment
article
breast epithelium
controlled study
enzyme activation
lactation
mouse
nonhuman
priority journal
protein expression
protein induction
animal
Bagg albino mouse
cell line
cytology
enzymology
gel mobility shift assay
human
immunohistochemistry
mammary gland
metabolism
physiology
Western blotting
Animals
Blotting, Western
Cell Line
Electrophoretic Mobility Shift Assay
Enzyme Activation
Humans
Immunohistochemistry
Leukemia Inhibitory Factor
Mammary Glands, Human
Mice
Mice, Inbred BALB C
Mitogen-Activated Protein Kinase 1
Mitogen-Activated Protein Kinase 3
Transcription Factor AP-1
Tumor Necrosis Factor-alpha
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_07302312_v110_n4_p857_Levy
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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