Chromatin remodeling and control of cell proliferation by progestins via cross talk of progesterone receptor with the estrogen receptors and kinase signaling pathways

Transcription from the mouse mammary tumor virus (MMTV) promoter can be induced by glucocorticoids or progestins. Progesterone treatment of cultured cells carrying an integrated single copy of an MMTV transgene leads to recruitment of progesterone receptor (PR), SWI/SNF, and SNF2h-related complexes...

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Detalles Bibliográficos
Autores principales:	Vicent, G.P., Ballaré, C., Zaurin, R., Saragüeta, P., Beato, M.
Formato:	SER
Materias:	Chromatin Histone H1 Kinases Mouse mammary tumor virus Progesterone receptor Transcriptional regulation adenosine triphosphate cell nucleus receptor DNA directed RNA polymerase III estrogen receptor gestagen heterochromatin protein 1 histone acetyltransferase PCAF histone H2A histone H2B histone H3 lysine mitogen activated protein kinase phosphotransferase progesterone progesterone receptor protein kinase B protein tyrosine kinase Ras protein regulator protein ribosome DNA steroid receptor protein kinase binding kinetics cell proliferation chromatin assembly and disassembly chromosome analysis concentration (parameters) conference paper cytoplasm DNA sequence endometrium cell enzyme activation enzyme activity enzyme phosphorylation gene sequence hormonal regulation human minichromosome mitosis inhibition molecular interaction Mouse mammary tumor oncovirus nonhuman nucleosome outcome assessment promoter region protein protein interaction protein purification signal transduction transcription regulation transgene animal drug effect gene expression regulation genetics metabolism mouse rat review Rattus Animals Cell Proliferation Chromatin Assembly and Disassembly Gene Expression Regulation Mammary Tumor Virus, Mouse Mice Progestins Promoter Regions (Genetics) Protein Kinases Rats Receptors, Estrogen Receptors, Progesterone Signal Transduction
Acceso en línea:	http://hdl.handle.net/20.500.12110/paper_00778923_v1089_n_p59_Vicent
Aporte de:	Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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