Hepatic arachidonic acid metabolism is disrupted after hexachlorobenzene treatment

Hexaclorobenzene (HCB), one of the most persistent environmental pollutants, can cause a wide range of toxic effects including cancer in animals, and hepatotoxicity and porphyria both in humans and animals. In the present study, liver microsomal cytochrome P450 (CYP)-dependent arachidonic acid (AA)...

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Detalles Bibliográficos
Autores principales:	Billi De Catabbi, S.C., Faletti, A., Fuentes, F., San Martín De Viale, L.C., Cochón, A.C.
Formato:	JOUR
Materias:	Arachidonic acid Cytochrome P450 Cytosolic phospholipase A2 Hexachlorobenzene Prostaglandin Rat liver aminopyrine n demethylase arachidonic acid cytochrome P450 ethoxyresorufin deethylase hexachlorobenzene phospholipase A2 prostaglandin E animal experiment animal model animal tissue arachidonic acid metabolism article clinical feature controlled study cytokine release enzyme activity female liver liver microsome liver toxicity membrane fluidity nonhuman porphyria cutanea tarda protein function rat statistical significance
Acceso en línea:	http://hdl.handle.net/20.500.12110/paper_0041008X_v204_n2_p187_BilliDeCatabbi
Aporte de:	Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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