Rol de las Heat Shock Proteins en tejido renal de ratas Zucker Diabetic Fatty

The aim of this study was to evaluate whether the "Heat Shock Proteins" (HSP) can control functional and morphological renal damage in a recognized animal model of type 2 diabetes (DMT2). Male 12-week old "Zucker Diabetic Fatty" (ZDF) rats and control "Lean Zucker Rat¨ (LZR)...

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Autor principal: Maselli, María del Carmen
Otros Autores: Angerosa, Margarita
Formato: Tesis doctoral acceptedVersion
Lenguaje:Español
Publicado: Facultad de Farmacia y Bioquímica 2018
Materias:
Rat
Acceso en línea:http://repositoriouba.sisbi.uba.ar/gsdl/cgi-bin/library.cgi?a=d&c=posgraafa&cl=CL1&d=HWA_2825
http://repositoriouba.sisbi.uba.ar/gsdl/collect/posgraafa/index/assoc/HWA_2825.dir/2825.PDF
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Sumario:The aim of this study was to evaluate whether the "Heat Shock Proteins" (HSP) can control functional and morphological renal damage in a recognized animal model of type 2 diabetes (DMT2). Male 12-week old "Zucker Diabetic Fatty" (ZDF) rats and control "Lean Zucker Rat¨ (LZR) were used. All animals received water and standard rat chow, "ad libitum". The immunoexpression of HSP70 and HSP27 was assessed in kidney tissue. Markers of glomerular and tubulointerstitial damage (oxidative stress, inflammation, nitrosative stress, fat deposition, apoptosis, interstitial fibrosis), and podocyte injury were analyzed. Ultrastructural glomerular alterations were evaluated by electron microscopy. We correlated biochemical variables and histological markers of podocyte damage and expression of HSPs in renal tissue. The conclusion of this study is that the increased expression of HSP70 and HSP27 found in kidney of ZDF rats, although it represents an activated repair mechanism in response to an adverse metabolic scenario, its participation does not succeed in modulating the injury caused in such tissue, showing insufficient or at least incomplete behavior due to the magnitude of renal cell damage induced in this experimental model of T2DM.