Resistance to cardiomyocyte hypertrophy in ae3-/- mice, deficient in the AE3 Cl-/HCO3- exchanger

Background: Cardiac hypertrophy is central to the etiology of heart failure. Understanding the molecular pathways promoting cardiac hypertrophy may identify new targets for therapeutic intervention. Sodium-proton exchanger (NHE1) activity and expression levels in the heart are elevated in many model...

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Autores principales: Sowah, D., Brown, B.F., Quon, A., Álvarez, Bernardo Víctor, Casey, J.R.
Formato: Articulo
Lenguaje:Inglés
Publicado: 2014
Materias:
AE3
Acceso en línea:http://sedici.unlp.edu.ar/handle/10915/85641
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