Advanced glycation end-products (AGEs) induce concerted changes in the osteoblastic expression of their receptor RAGE and in the activation of extracellular signal-regulated kinases (ERK)

An increase in the interaction between advanced glycation end-products (AGEs) and their receptor RAGE is believed to contribute to the pathogenesis of chronic complications of Diabetes mellitus, which can include bone alterations such as osteopenia. We have recently found that extracellular AGEs can...

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Detalles Bibliográficos
Autores principales: Cortizo, Ana María, Lettieri, María Gabriela, Barrio, Daniel Alejandro, Mercer, Natalia, Etcheverry, Susana B., McCarthy, Antonio Desmond
Formato: Articulo Preprint
Lenguaje:Inglés
Publicado: 2003
Materias:
Ciencias Exactas
advanced glycation end-products, receptor for advanced glycation end-products, extracellular signal-regulated kinases, osteoblasts, bone
quinasas
Acceso en línea:http://sedici.unlp.edu.ar/handle/10915/67404
https://link.springer.com/article/10.1023%2FA%3A1024934008982
Aporte de:
SEDICI (UNLP) de Universidad Nacional de La Plata

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