AMPK-dependent nitric oxide release provides contractile support during hyperosmotic stress

In different pathological situations, cardiac cells undergo hyperosmotic stress (HS) and cell shrinkage. This change in cellular volume has been associated with contractile dysfunction and cell death. Given that nitric oxide (NO) is a well-recognized modulator of cardiac contractility and cell survi...

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Autores principales: Morell, Malena, Burgos Migone, Juan Ignacio, Gonano, Luis Alberto, Vila Petroff, Martín Gerardo
Formato: Articulo
Lenguaje:Inglés
Publicado: 2018
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Acceso en línea:http://sedici.unlp.edu.ar/handle/10915/136191
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