Reversible redox modifications of ryanodine receptor ameliorate ventricular arrhythmias in the ischemic-reperfused heart

Previous results from our laboratory showed that phosphorylation of ryanodine receptor 2 (RyR2) by Ca21 calmodulin-dependent kinase II (CaMKII) was a critical but not the unique event responsible for the production of reperfusion-induced arrhythmogenesis, suggesting the existence of other mechanisms...

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Detalles Bibliográficos
Autores principales: Becerra, Romina Valeria, Román, Bárbara Soledad, Di Carlo, Mariano Nahuel, Mariángelo, Juan Ignacio Elio, Salas, Margarita Ana, Sánchez, Gina, Donoso, Paulina, Schinella, Guillermo Raúl, Vittone, Leticia, Wehrens, Xander H., Mundiña-Weilenmann, Cecilia, Said, María Matilde
Formato: Articulo
Lenguaje:Inglés
Publicado: 2016
Materias:
Ciencias Médicas
arrhythmias
ischemia/reperfusion
ryanodine receptor type 2
redox modifications
Acceso en línea:http://sedici.unlp.edu.ar/handle/10915/103856
Aporte de:
SEDICI (UNLP) de Universidad Nacional de La Plata

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