Death of adrenocortical cells during murine acute T. cruzi infection is not associated with TNF-R1 signaling but mostly with the type II pathway of Fas-mediated apoptosis
Earlier studies from our laboratory demonstrated that acute experimental Trypanosoma cruzi infection promotes an intense inflammation along with a sepsis-like dysregulated adrenal response characterized by normal levels of ACTH with raised glucocorticoid secretion. Inflammation was also known to res...
Guardado en:
| Autores principales: | Pérez, Ana Rosa, Lambertucci, Flavia, González, Florencia Belén, Roggero, Eduardo Angel, Bottasso, Oscar, de Meis, Juliana, Ronco, Maria Teresa, Villar, Silvina Raquel |
|---|---|
| Formato: | article artículo acceptedVersion |
| Lenguaje: | Inglés |
| Publicado: |
Academic Press Inc Elsevier Science
2019
|
| Materias: | |
| Acceso en línea: | http://hdl.handle.net/2133/14298 http://hdl.handle.net/2133/14298 |
| Aporte de: |
Ejemplares similares
-
Molecular and functional evidence for in vitro cytokine enhancement of human and murine target cell sensitivity to glucocorticoids: TNF-α priming increases glucocorticoid inhibition of TNF-α-induced cytotoxicity/apoptosis
por: Costas, M., et al.
Publicado: (1996) -
Molecular and functional evidence for in vitro cytokine enhancement of human and murine target cell sensitivity to glucocorticoids: TNF-α priming increases glucocorticoid inhibition of TNF-α-induced cytotoxicity/apoptosis
por: Costas, M., et al.
Publicado: (1996) -
Molecular and functional evidence for in vitro cytokine enhancement of human and murine target cell sensitivity to glucocorticoids: TNF-α priming increases glucocorticoid inhibition of TNF-α-induced cytotoxicity/apoptosis
por: Costas, M., et al. -
Molecular and functional evidence for in vitro cytokine enhancement of human and murine target cell sensitivity to glucocorticoids: TNF-α priming increases glucocorticoid inhibition of TNF-α-induced cytotoxicity/apoptosis
por: Costas, Mónica Alejandra, et al.
Publicado: (1996) -
Transrepression of NF-κB is not required for glucocorticoid-mediated protection of TNF-α-induced apoptosis on fibroblasts
por: Costas, M.A., et al.
Publicado: (2000)